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Although choline, betaine and carnitine can predict the risk of future MACE, this association appears to be primarily driven by the gut microbiota-derived metabolite TMAO [3, 4]

Under normal conditions, Nrf2 is constantly ubiquitinated through Keap1 and degraded in the proteasome

As a result, the methylation cycle will be compromised or impaired, along with many other functions that depend on it, including glutathione production

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