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Defective decidualization during and after severe preeclampsia reveals a possible maternal contribution to the etiology
The study included 36 PCD patients, 17 carriers and 17 healthy subjects
Activation transpires via the alteration of Keap1 cysteine residues, hence breaking the Keap1-Nrf2 complex and facilitating Nrf2s translocation to the nucleus
renal impairment may require monitoring The one-week half-life of both components enables true once-weekly dosing while maintaining relatively stable plasma concentrations throughout the dosing interval, contributing to consistent therapeutic effects and potentially improved tolerability compared to shorter-acting agents
This reaction is catalyzed by glutathione with the simultaneous formation of GSSG [13]
doi:10.3389/fgene.2021.652704
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