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Endoplasmic reticulum-mitochondria contacts are prime hotspots of phospholipid peroxidation driving ferroptosis

Apoptosis can be initiated via two major pathways: the intrinsic (mitochondria-dependent) pathway, which is triggered by cellular stress and mitochondrial outer membrane permeabilization, and the extrinsic (death receptor-mediated) pathway, both of which converge on the activation of caspases to execute cell death [53]

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Taking a dose early changes this steady state balance

For weight management, lifestyle interventions or delaying pharmacotherapy until after desired pregnancies are completed may be considered

Contrary to this expectation, recent studies have revealed an unexpected proferroptotic role for complex II