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GHK-Cu acts at the extracellular matrix (ECM) level, modulating the balance between matrix metalloproteinases (MMPs) and their inhibitors (TIMPs) to regulate collagen turnover, and stimulating fibroblasts to produce collagen type I, collagen type III, and elastin
Frontiers in Physiology, 14 , 1149120
Bojanic, Ivan
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This research has implications for understanding the regulation of NAD+-dependent processes in cells
But hidden within those basic steps are dozens of opportunities to damage or degrade the copper peptide complex before it ever reaches your skin or tissue
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