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Not only does it regulate the balance between NSC self-renewal and differentiation but it also directly affects the survival of newly generated neurons ( In vitro experiments have demonstrated that hypoxic conditions enhance the stability of -catenin and the expression of its downstream target genes (Figure 1), thereby promoting NSC proliferation, increasing survival rates, and enhancing differentiation potential ( However, the effects of hypoxia on NSCs are evidently concentration- and time-dependent

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