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Our study showed that HepG2 cells, which primarily rely on extracellular cystine as a source of cysteine 20 , responded to cystine deprivation by reducing anabolic signaling including attenuation of mTORC1 pathway and activation of the ISR (Fig

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Originally developed to treat HIV-associated lipodystrophy, it has shown impressive results in metabolic syndrome and general abdominal obesity

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Plasma concentrations return to baseline within 12 hours after intravenous administration of 0.5 g [5][12]

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3, June 2005, pp

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