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, Mediator effects of parameters of inflammation and neurogenesis from a N-acetyl cysteine clinical-trial for bipolar depression, Acta Neuropsychiatr

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Neuroinflammation is primarily mediated by activated microglia and astrocytes that release pro-inflammatory cytokines, thereby exacerbating central nervous system (CNS) inflammation (Ahmad et al

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When glutathione stores are depleted, the liver becomes more vulnerable to the toxic effects of acetaminophen