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The pharmacology and neurotoxicology of synthetic cathinones

NAD(+) improves cognitive function and reduces neuroinflammation by ameliorating mitochondrial damage and decreasing ROS production in chronic cerebral hypoperfusion models through Sirt1/PGC-1alpha pathway

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Flagg EW, Coates RJ, Eley JW, Jones DP, Gunter EW, Byers TE et al

After donating electrons, it becomes oxidized and is then recycled back to GSH by glutathione reductase using NADPH, creating a continuous antioxidant recharge loop that supports day-to-day cellular resilience