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Without this offset, both compounds compete for limited NAD+ and receptor access simultaneously, creating a metabolic bottleneck that reduces efficacy of both

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Pro-inflammatory cytokines (such as IL-1) and oxidative stress factors (such as the oxidant TBHP) are frequently utilized as inducers to establish OA models in chondrocytes, and a reduction in GPX4 expression is typically observed in these treated cells [11,65]

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Example calculation: (5 10) 100 = 50 units