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By continuing to explore the intricate connections between glutathione, redox signaling, and autism, researchers aim to develop targeted interventions and treatments that address the underlying mechanisms of autism and improve the quality of life for individuals affected by this condition

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This review aims to highlight the central role of OS in the pathophysiology of ALS, specifically focusing on the KEAP1-NRF2 system and its alteration in ALS, while discussing the potential applications of NRF2 activators in ALS therapy

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