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we leveraged a unique model of GLP-1R deficiency, Glp1r Wnt1-/- mice, which are resistant to GLP-1RA-induced weight loss
Improved erythrocyte survival with high dose vitamin E in chronic hemolysing G6PD and glutathione synthetase deficiency
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Therefore, we hypothesized that the excessive expression of GJs post-APAP exposure in hepsin / mice could be a key factor contributing to the increased severity of liver damage observed following APAP administration
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