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Current surveillance has not demonstrated a substantial rise in cases linked to GLP-1 therapies

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Mitochondrial ROS production and oxidative mtDNA damage is increased during reoxygenation in the GPX1 knockout mice, in addition to structural abnormalities in cardiac mitochondria and myocytes, suggesting GPX1 may play an important role in protecting cardiac mitochondria from reoxygenation damage in vivo

Therapeutic interventions aimed at reducing protein oxidation typically focus on antioxidant supplementation, which has been shown to improve sperm motility and reduce oxidative damage in clinical studies

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PTX is generally well-tolerated, but some patients experience adverse effects, primarily gastrointestinal disturbances such as nausea, vomiting, dyspepsia, and bloating [30]