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Unlike GSH, supplementation with these antioxidants did not supply cysteine, which suggests that preventing lipid peroxidation after cystine deprivation is not sufficient to sustain cancer cells
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Glutathione may be one such supplement
Otherwise process has been easy
As superoxide is subject to efficient dismutation by cellular superoxide dismutases, it is possible that NFT might directly interact with Keap1 to induce the observed release of Nrf2 and its escape from proteasomal degradation
The acute augmentation of tumor cells consumes more nutrients than normal cells, so the metabolism of tumor cells requires a high consumption of oxygen and glucose [51]
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