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This review synthesizes current evidence on the molecular and clinical mechanisms of atorvastatin-induced hepatotoxicity, emphasizing genetic susceptibility, transporter interactions, and mitochondrial dysfunction

Rodents overexpressing USP22 or lncRNA PART1 were well resistant to I/R-induced oxidative stress and cardiac injury, which may be partly attributed to the elevated GSH content in the myocardium [251, 252]

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L-Carnitine moiety assay: an up-to-date reappraisal covering the commonest methods for various applications

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