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Although canonically GLP1Rs signal through the stimulatory alpha Gs protein, we find that semaglutide utilizes both Gs- and Gq-signaling pathways in GLP1Rs in the area postrema (AP)the primary site of semaglutide action in the brainto differentially regulate neuronal activation across distinct neuronal clusters
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As fitting function, the following equation was used where M z (t) is the longitudinal relaxation at the recovery delay time t , M 0 is the equilibrium magnetization and R i is either the longitudinal relaxation rate R 1 or R 1
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