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In white adipose tissue of obese, insulin-resistant and aged mammals, NNMT is dramatically upregulated and because it consumes both the universal methyl donor (SAM) and the NAD precursor (nicotinamide), its hyperactivity simultaneously depletes the cellular methylation reserve and the NAD-salvage pool [1,2]
Its refreshing to deal with a company that clearly values its customers, as I had many questions prior to purchase
doi:10.1076/opep.10.4.215.15906
PMC 4575440
Research Use Cases: Research shows AOD 9604 is primarily utilized in studies focusing on weight management and metabolic enhancement
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