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Activation of G-protein-coupled receptors, such as luteinizing hormone and follicle-stimulating hormone receptors, may be the molecular underpinning of the signs and symptoms of the syndrome
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Importantly, increasing the endogenous production of GLP-1 by different mechanisms or by increasing the number of intestinal L-cells that tend to produce this hormone may be another effective therapeutic approach to managing T2DM
In support to this theory, it has been demonstrated that GIP infusion increases the proliferation of neuronal progenitors in the dentate gyrus, while mice lacking GIP displayed deterioration in memorial tasks as a result of reduced neurogenesis [24]
