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Moreover, BCA was shown to impede chondrocyte ferroptosis via modulating iron homeostasis, diminishing ROS buildup, and activating the Nrf2/system XC-/GPX4 signaling pathway, which combined facilitated its chondroprotective effects [102]

These findings provide a comprehensive description of OVX-induced glutathione redox stress at multiple levels and support HPL therapy as an effective modulator in renal tissues to locally influence the glutathione metabolism pathway and subsequent redox homeostasis

Several therapeutic strategies exploit this pathway

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Its described antinociceptive effect in animals might decrease pain-induced impotence and increase sexual and reproductive function