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The upsurge of very-low-density lipoprotein particles seems to be the core atherogenic dyslipidemia in T2DM, which is partially secondary to insulin resistance [26]

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But it also comes with an important caveat that isnt getting enough attention: this regulatory change is not the same as FDA approval, and how you access these therapies matters just as much as whether you can

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DSIP was less potent than GnRH and as potent as TRH in inducing LH and TSH differentiation