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The retina and CNS share susceptibility to mitochondrial dysfunction, redox imbalance, and inflammation, although the retina constitutes a highly specialized neural tissue rather than a direct model of CNS neurodegeneration, with distinct metabolic demands, cellular architecture, and stressors that shape disease mechanisms and therapeutic responsiveness

This does not effect the efficacy of the product, only the texture

Also, it produces serotonin in the body

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This creates a dual problem: the meal stays in your system longer, and the FFAs themselves act as a brake on somatotroph function [6]

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