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Wagner AM, Miranda-Calderin G, Ugarte-Lopetegui MA, Marrero-Santiago H, Suarez-Castellano L, Lopez-Madrazo MJ, et al

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The remarkable diversity of these mechanisms reflects both the complex, redundant nature of inflammatory pathways in UC and the recognition that single-target approaches may have inherent efficacy limitations, potentially necessitating either highly selective patient stratification based on dominant pathogenic mechanisms or rational combination strategies targeting complementary pathways

Antioxidative Aktivitt GHK-Cu strkt die endogenen antioxidativen Abwehrkrfte durch Steigerung der Expression von Superoxiddismutase (SOD), Glutathionperoxidase und Ferritin