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Among these, autosomal dominant forms include mutations in SNCA (PARK1), UCHL1 , LRRK2 (PARK8), CHCHD2 , GBA , and VPS35 (PARK17), whereas autosomal recessive forms involve PARK2 (parkin), PINK1 (PARK6), DJ-1 (PARK7), and ATP13A2 (PARK9) (260)

The SECIS selenocysteine (sec) insertion sequence represents mRNA and serves as a platform for the recruitment of Sec -ARNt translation stretching factors that decode the UGA codon for the incorporation of Se into selenoproteins [43]

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