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Alterations in the normal function of excitatory amino acid carrier 1 affect cysteine transport and GSH synthesis impairing zinc homeostasis (the thiol group of GSH can function as a principal Zn 2+ chelator for the maintenance of Zn 2+ homeostasis in neurons) and oxidative stress, enhancing susceptibility to ischemia-induced neuronal cell death in the hippocampus (162164)

Recently, several advances highlight its novel involvement in ferroptosis, an iron-dependent programmed cell death mechanism characterized with lipid peroxidation and iron accumulation

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Injections of glutathione prevent oxidative stress that the body experiences as a result of an excess of free radicals