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As an aside, piperlongumine has an effect on restraining FTC cell growth and cell cycle, inducing apoptosis and autophagy by PI3K/AKT pathway [292]

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Although N-acetylcysteine (NAC) directly influences the pool of extracellular cystine and intracellular cysteine via a series of plasmatic redox reactions, in order to be effective, intracellular cysteine precursors must be designed to enter erythrocytes rapidly and use high activity enzymes within erythrocytes to liberate cysteine (Whillier et al., 2009

TMAO has been implicated in promoting inflammation and has been associated with increased risk of stroke and cognitive impairment [44, 49]