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This review aims to summarize current evidence on the mechanisms by which oxidative stress and metabolic abnormalities are associated with impaired endometrial receptivity in PCOS, with a particular focus on the emerging role of microbiota as an integrative upstream regulator linking metabolic dysfunction, inflammatory activation, and endometrial impairment

Failure of DNA repair induces apoptosis through p53 activation, mitochondrial pathway induction, and persistent DNA damage response (DDR) signaling 8-10

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The approach uses adeno-associated virus vectors to deliver the BDNF gene directly into the brain, enabling continuous local production of the protein

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