Both operate through distinct mechanisms: GLP-1 agonism enhances insulin secretion and slows gastric motility, while glutathione functions as a cellular antioxidant without direct endocrine effects
2016 Jan;46(1):35-47
In the unlikely event of significant copper toxicity, treatment involves IV chelation therapy using EDTA, which binds to excess copper and allows it to be excreted through the kidneys
Beyond glomerular damage, tubular atrophy and tubulointerstitial inflammation have also been demonstrated, while vascular lesions include arterial hyalinosis [16]
et al., Bioorg Med Chem Lett, 2015
However, GHK-Cu goes further by: Stimulating collagen more directly Enhancing tissue repair Reducing inflammation Vitamin C and GHK-Cu serve different purposes, so combination therapy with both usually works better