The effects of the mutation are reduced by treatment with NACA, resulting in increased survival of Schwann cells
This means that even when fractures are not present, our bones are in a state of perpetual remodeling
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While LipRSV reduced oxidative markers in the hippocampus, cerebellum and posterior cortex, the increase observed in the frontal cortex suggests that nanocarrier distribution and local redox balance may critically influence therapeutic outcomes