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However, 3T3-L1 preadipocytes treated with LA exhibit lower insulin-induced differentiation by modulating activity and/or expression of various anti-adipogenic transcription factors mainly through activating the MAPK pathways that negatively regulate PPAR and C/EBP [141]

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Importantly, these expanded HBs retained bipotency, co-expressing both hepatic and early cholangiocyte markers [1]

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