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In particular, the upregulation of IL6, IL1, and Stat3, along with alterations in immune cell populations such as MDSCs and regulatory T cells, and inflammasome suggest plausible mechanistic links to BC pathogenesis 137
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CD8 + cytotoxic T lymphocytes, reactive to TPO and Tg, promote glandular destruction, aided by increased ICAM-1 expression on follicular cells and elevated serum levels of VCAM-1 and TIMP-1
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