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At present, the therapies of Alzheimers disease include stem cell therapy, gene therapy, and chemotherapy, but these therapies have limitations inevitably such as inefficient and neurotoxicity (Jiang et al., 2015b

Furthermore, its protective effect in APAP-induced hepatotoxicity was linked to reduced mitochondrial dysfunction, oxygen consumption, and membrane potential (Lu et al., 2015

This aligns with the well-established understanding that uremia and dialysis procedures promote excessive reactive oxygen species generation (4)

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