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It is essential to create predictive animal models, primary human tissue culture systems, and virtualized medications based on particular genetic profiles of fibrotic patients due to the developments of modern technology, such as single-cell multi-omics

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However, such impairments in components of mitochondrial metabolism may not directly translate to alterations of metabolic fluxes in vivo due to potential compensatory mechanisms

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Setting realistic expectations prevents discouragement during the initial phase when progress may not yet be visible