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Assessment of cardiovascular risk factors and kidney function, which may affect dosing

Although our analyses show that many of the proteomic perturbations resulting from semaglutide treatment are linked to changes in body weight and glycemia, the lack of a dose response on CVD risk with semaglutide 1.0 mg and 2.4 mg in STEP 2 supports semaglutide effects on CVD beyond weight loss

Investigational evidence base: Studied in clinical research for effects on body weight, energy intake, and metabolic markers in overweight/obesity populations

Clinical trials have shown meaningful reductions in A1c levels (a marker of average blood sugar) in people taking semaglutide

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124,125,126 The design concept of these multi-agonists is to simultaneously regulate multiple key metabolic pathways to achieve more effective control over blood sugar, body weight, and overall metabolic health