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This oxidative stress is particularly damaging because of METHs effects on dopamine, which undergoes autoxidation, leading to the generation of dopamine quinones and ROS (Figure 1)
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In vitro reversal of glutathione-S-transferase-mediated resistance in canine osteosarcoma (COS31) cells
FIGURE 1 The phosphoinositide 3-kinase-Akt-mammalian target of rapamycin (PI3K-Akt-mTOR) signaling pathway, commonly activated in cancer, including AML, contributes to the maintenance of glycolysis by translational and post-translational regulation of metabolic enzymes (Hoxhaj and Manning, 2020
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