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(33) demonstrated that transient receptor potential melastatin 4 (TRPM4) channels significantly contribute to oxidized low-density lipoprotein (ox-LDL)-induced dysfunction in human coronary artery endothelial cells (HCAECs)
Results were achieved as part of a provider-directed GLP-1 treatment program
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doi:10.1186/1752-1947-2-29
When cells migrate to a wound site or form new tissue, actin plays a central role in making that movement possible [2]
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